Do you need this or any other assignment done for you from scratch?
We have qualified writers to help you.
We assure you a quality paper that is 100% free from plagiarism and AI.
You can choose either format of your choice ( Apa, Mla, Havard, Chicago, or any other)
NB: We do not resell your papers. Upon ordering, we do an original paper exclusively for you.
NB: All your data is kept safe from the public.
What is occurring in the patient?
Weakness, visual disturbances, fatigue, and nausea are complications that indicate about two medical issues. The first indication that requires diagnosis is the side effects associated with the recent prescription of Lisinopril. It is important to explain that Lisinopril has side effects that comprise weakness, nausea, and headaches. As such, by presenting these complications, Elliot could be experiencing the side effects of Lisinopril. The second diagnosis that requires attention is a condition known as hypokalemia, which transpires due to low levels of potassium. When the levels of potassium in the blood fall below 3.5mEq/liter, the person is likely to suffer from the complications presented by Elliot. Allan, Ivers, and Padwal (2012) allude that when one uses drugs like Thiazide and Digoxin, there is a high probability that the drugs will inhibit the overall secretion and utility of potassium in the body. Therefore, the level of potassium drops below the optimum scale and triggers hypokalemia.
In the context of Lisinopril, the precise mechanisms that lead to the complications presented by Elliot revolve around the processes initiated by the medication. When the drug inhibits the conversion of angiotensin from I to II, it impairs the functionality of vasoconstriction. Some of the side effects that develop after using Lisinopril, which include coughs and weakness, take place after the drug triggers hypotension, a scenario characterized by the low pressure of blood. The diastolic and systolic pressure of blood drops below therapeutic levels and leads to an eventuality of the issues presented by Elliot. Consequently, the scenario that relates to low levels of potassium transpires when Digoxin and Thiazide interact and initiate increased secretion of sodium in the body. In the assertion of Speer (2016), Digoxin is responsible for triggering a sodium overload through its role of inhibiting the processes of myocardial cells. Additionally, the drug raises the amount of sarcoplasmic calcium, a factor that lowers the overall levels of potassium in the body.
Fundamentally, potassium is a very important mineral as it maintains the electoral activities of the cells. Therefore, when Digoxin hampers the functionality of the mineral, the person can present issues evidenced by Elliot. Sostaric (2012) explains that while Digoxin is responsible for sodium and calcium overloads, Hydrochlorothiazide facilitates increased secretion of calcium, hydrogen, chloride, magnesium, and potassium. The increased secretion of potassium implies that its levels in the body diminish because it is utilized after secretion. Besides initiating increased secretion of potassium, Thiazide also prevents the effects of the distal tubule in reabsorbing sodium. Notably, increased secretion of potassium initiated by Thiazide and inhibited re-absorption of sodium is one of the factors that minimize the levels of potassium and amplify the amount of sodium. The implication of the low amount of potassium is hypokalemia associated with the problems experienced by Elliot.
Drug interactions associated with any new medications initiated keeping in mind the current regimen
The current regimen of drugs used in addressing Elliot’s case displays a high probability of interaction. Drugs such as Thiazide and Digoxin can interact because of their roles in the body. The interaction can lead to increased sodium in the body, hypokalemia, as well as Digoxin toxicity. As such, by prescribing Thiazide together with Digoxin in a single regimen, the symptoms presented by Elliot are likely to occur. Sostaric (2012) highlights the fact that a combination of Thiazide and Digoxin leads to low amounts of potassium in the blood and affects homeostatic processes. The explanation substantiates the interaction that exists between Thiazide and Digoxin and the likely complications.
References
Allan, G., Ivers, N., & Padwal, R. (2012). Best thiazide diuretic for hypertension. Canadian Family Physician, 58(6), 653-653.
Sostaric, S. (2012). Alkalosis and digoxin effects on plasma potassium, ionic homeostasis and exercise performance in healthy humans. Melbourne, Australia: Victoria University.
Speer, B. (2016). Current therapy in avian medicine and surgery. St. Louis, MO: Elsevier.
Do you need this or any other assignment done for you from scratch?
We have qualified writers to help you.
We assure you a quality paper that is 100% free from plagiarism and AI.
You can choose either format of your choice ( Apa, Mla, Havard, Chicago, or any other)
NB: We do not resell your papers. Upon ordering, we do an original paper exclusively for you.
NB: All your data is kept safe from the public.